Fas Ligand (FASL) (Biotin)

Supplier:

United States Biological

Cat no: F0019-66S

Fas Ligand (FASL) (Biotin)

Application(s): \nFlow Cytometry\nImmunocytochemistry\nWestern Blot\n\nFor immunohistochemistry, we recommend MAb to FasL (A11). For the detection of human FasL by flow cytometry or Western blot, we recommmend MAb to FasL (human) (Mike-1) and MAb to FasL (human) (Mike-2).

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SPECIFICATIONS

Catalog Number

F0019-66S

Size

100Tests

Applications

FC, ICC, IHC, WB

Hosts

Rat

Reactivities

Mouse

Conjugates

Biotin

Form

Liquid. Purified antibody in PBS containing 0.02% sodium azide.\n

P Type

Mab

Purity

(same/more than)95% (by SDS-PAGE).

Isotype

IgG2a

References

Product Specific Literature References\n1. Characterization of the non-functional Fas ligand of gld mice: M. Hahne, et al.; Int. Immunol. 7: 1381 (1995). 2. CD4+ T cells reactivated with superantigen are both more sensitive to FasL-mediated killing and express a higher level of FasL: J.K. Wang, et al.; Cell. Immunol. 179: 153 (1997). 3. Transgenic expression of CD95 ligand on thyroid follicular cells confers immune privilege upon thyroid allografts: L. Tourneur, et al.; J. Immunol. 167: 1338 (2001). \nGENERAL LITERATURE REFERENCES\n1. Mature T cells of autoimmune lpr/lpr mice have a defect in antigen-stimulated suicide: H. Russell, et al.; PNAS 90: 4409 (1993), 2. Cytolytic T-cell cytotoxicity is mediated through perforin and Fas lytic pathways: B. Lowin, et al.; Nature 370: 650 (1994). 3. A role for CD95 ligand in preventing graft rejection: D. Bellgrau, et al.; Nature 377: 630 (1995). 4. Fas ligand-induced apoptosis as a mechanism of immune privilege: T.S. Griffith, et al.; Science 270: 1189 (1995). 5. Apoptosis by death factor: S. Nagata; Cell 88: 355 (1997).

Additional Info

Recognizes mouse FasL.

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SUPPLIER INFO

United States Biological

Address:

4 Technology Way, Salem, MA 01970, US

Phone:

+1 800 520 3011

Website:

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