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GSS pAb

GSS pAb

Cat no: BS6121


Supplier: Bioworld Technology, Inc.
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Defects in GSS are the cause of glutathione synthetase deficiency (GSS deficiency); also known as 5-oxoprolinuria or pyroglutamic aciduria. It is a severe form characterized by an increased rate of hemolysis and defective function of the central nervous system. Defects in GSS are the cause of glutathione synthetase deficiency of erythrocytes (GLUSYNDE). Glutathione synthetase deficiency of erythrocytes is a mild form causing hemolytic anemia.
Catalogue number: BS6121
Reactivities: Human, Mouse, Rat
Hosts: Rabbit
Applications: Immunohistochemistry, Western Blot
Size: 100ug/100ul
Swiss prot: P48637
Dilutions: WB: 1:500~1:2000 IHC: 1:50~1:200
Form: liquid
Storage buffer: 1 mg/ml in Phosphate buffered saline (PBS) with 0.05% sodium azide, approx. pH 7.2.
Concentration: 1ug/ul
Antigen: Recombinant full length Human GSS.
Species: GSS pAb detects endogenous levels of GSS protein.
Molecular weight: ~ 52 kDa
Purity: The antibody was affinity-purified from rabbit antiserum by affinity-chromatography using epitope-specific immunogen and the purity is > 95% (by SDS-PAGE).
Storage temp: Store at 4 degrees C short term. Aliquot and store at -20 degrees C long term. Avoid freeze-thaw cycles.
Alternative names: Glutathione synthase; Glutathione synthetase; GSH S; GSH synthetase; GSH-S; GSHB; GSHS; GSS; MGC14098; OTTHUMP00000030711;
Additional info: For research use only, not for use in diagnostic procedure.

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